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16号染色体开放阅读框7抗体
AK18471
C16orf7
货号 规格 目录价 会员价 库存 数量 购买
AK18471-50ul 50ul ¥1380.00 登录查看
AK18471-100ul 100ul ¥2380.00 登录查看
AK18471-200ul 200ul ¥3480.00 登录查看
产品详情

公司产品仅供科研研究实验,不得用于临床!

商品详情:

英文名称:C16orf7

中文名称:16号染色体开放阅读框7抗体

    名;ATP BL; C16orf7; Chromosome 16 open reading frame 7; CP007_HUMAN; Protein ATP-BL; Uncharacterized protein C16orf7.

研究领域;肿瘤  细胞生物  免疫学  神经生物学

抗体来源;Rabbit

克隆类型;Polyclonal

交叉反应;Human, Rat,  (predicted: Mouse, Cow, )

产品应用;WB=1:500-2000 ELISA=1:5000-10000 IHC-P=1:100-500 IHC-F=1:100-500 ICC=1:100-500 IF=1:100-500 (石蜡切片需做抗原修复)

not yet tested in other applications.

optimal dilutions/concentrations should be determined by the end user.

理论分子量;69kDa

细胞定位;细胞核 细胞浆

    状;Liquid

    度;1mg/ml

免 疫 原;KLH conjugated synthetic peptide derived from human C16orf7/ATP-BL : 25-130/631

    型;IgG

纯化方法;affinity purified by Protein A

缓 冲 液;0.01M TBS(pH7.4) with 1% BSA, 0.03% Proclin300 and 50% Glycerol.

保存条件;Shipped at 4. Store at -20 °C for one year. Avoid repeated freeze/thaw cycles.

注意事项;This product as supplied is intended for research use only, not for use in human, therapeutic or diagnostic applications.

产品介绍;Chromosome 16 encodes over 900 genes in approximately 90 million base pairs, makes up nearly 3% of human cellular DNA and is associated with a variety of genetic disorders. The GAN gene is located on chromosome 16 and, with mutation, may lead to giant axonal neuropathy, a nervous system disorder characterized by increasing malfunction with growth. The rare disorder Rubinstein-Taybi syndrome is also associated with chromosome 16, though through the CREBBP gene which encodes a critical CREB binding protein. Signs of Rubinstein-Taybi include mental retardation and predisposition to tumor growth and white blood cell neoplasias. Crohn's disease is a gastrointestinal inflammatory condition associated with chromosome 16 through the NOD2 gene. An association with systemic lupus erythematosis and a number of other autoimmune disorders with the pericentromeric region of chromosome 16 has led to the identification of SLC5A11 as a potential autoimmune modifier.

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